Local hypodynamic osteoresorption: medical and social causes and pathogenetic mechanisms
Автор: Artemenkov A.A.
Журнал: Международный журнал гуманитарных и естественных наук @intjournal
Рубрика: Медицинские науки
Статья в выпуске: 5-1 (56), 2021 года.
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The aim of this study was to identify risk factors for a decrease in bone mineralization in the population to ensure the prevention of osteopenic conditions and osteoporosis. Two methods of investigation were used in the work: questioning and x-ray osteodensitometry. The survey involved 200 students aged 17-19 years. And with the help of X-ray osteodensitometry, the adult female population of mature, elderly and senile age in the number of 100 people (25 people in each age group) was examined. The object of the study was the distal epiphyses of the forearm bones and heel bone. In this paper, it is shown that the main risk factor for the development of osteopenic conditions in adolescents is the rare intake of dairy products and fish as sources of easily digestible calcium and phosphorus. We determined that the most frequently fractured are the phalanges of the fingers, the radial and ulnar bones of the forearm. It was found out that the mineral bone density in females in elderly and senile age is significantly lower in the distal epiphyses of the bones of the forearm than in the calcaneus. It has been suggested that a decrease in bone mineral density is associated with a lower stato- dynamic and mechanical load on the forearm bones than on the bones of the foot. To explain the pathogenetic mechanisms of destructive changes in some areas of bone tissue, the term "local hypodynamic bone loss" was introduced. The conclusion is made about the need for a balanced diet for the normalization of phosphoric-calcium metabolism, the use of physical exercises to strengthen the musculoskeletal apparatus of the distal sections of the upper limbs.
Mineral bone density, osteopenia, osteoporosis, risk factors, bone fractures, densitometry, prevention
Короткий адрес: https://sciup.org/170188824
IDR: 170188824 | DOI: 10.24412/2500-1000-2021-5-1-144-150