Role of chronic inflammation of the uterine mucosa in infertility (review of literature)

It is proved that the defeat of the uterine mucosa in ChE is accompanied by receptor inferiority, a change in the microvasculature and fibrotic changes at the site of the lesion that cause uterine ischemia, and this in turn provokes angiogenesis and sclerosis. The long-term effect of antigens ends with its functional overstrain depletion and progression of autoimmune processes, even more negatively affecting the tissues.