Effect of slow coronary flow on signal-averaged electrocardiogram

Автор: Nough Hossein, Khoshnood Elahe Rafie, Naghedi Aryan, Hadiani Leila, Vahid Jorat Mohammad

Журнал: Cardiometry @cardiometry

Рубрика: Original research

Статья в выпуске: 13, 2018 года.

Бесплатный доступ

The slow flow coronary or decreased coronary TIMI flow rate is characterized by delayed pacification of coronary vessels in the absence of any evidence of obstruction coronary artery disease and is detected by coronary angiography. In the present study, we aimed to evaluate the effects of slow coronary artery flow on signal-averaged electrocardiogram (SAECG) as a possible indicator of increased risk for ventricular arrhythmias and sudden cardiac death.

Late potential, slow coronary flow, signal-averaged electrocardiogram, sudden cardiac death, ventricular arrhythmias

Короткий адрес: https://sciup.org/148308851

IDR: 148308851   |   DOI: 10.12710/cardiometry.2018.13.4247

Список литературы Effect of slow coronary flow on signal-averaged electrocardiogram

  • Tambe A, et al. Angina pectoris and slow flow velocity of dye in coronary arteries-a new angiographic finding. American heart journal. 1972;84(1):66-71.
  • Singh S, Kothari S, Bahl V. Coronary slow flow phenomenon: an angiographic curiosity. Indian Heart J. 2004;56(6):613-7.
  • Diver DJ, et al. Clinical and arteriographic characterization of patients with unstable angina without critical coronary arterial narrowing (from the TIMI-IIIA Trial). The American journal of cardiology. 1994;74(6):531-7
  • Mangieri E, et al. Slow coronary flow: clinical and histopathological features in patients with otherwise normal epicardial coronary arteries. Catheterization and cardiovascular diagnosis. 1996;37(4):375-81
  • Mosseri M, et al. Histologic evidence for small-vessel coronary artery disease in patients with angina pectoris and patent large coronary arteries. Circulation. 1986;74(5): 964-72.
  • Sarıkaya S, et al. Assesment of Autonomic Function in Slow Coronary Flow using Heart Rate Variability and Heart Rate Turbulence. Angiol. 2013;2(117):2.
  • Camsarl A, et al. Endothelin-1 and nitric oxide concentrations and their response to exercise in patients with slow coronary flow. Circulation journal: official journal of the Japanese Circulation Society. 2003;67(12):1022-8.
  • Yuksel S, et al. Abnormal nail fold capillaroscopic findings in patients with coronary slow flow phenomenon. Int J Clin Exp Med. 2014;7(4):1052-8.
  • Demir M, Melek M. Evaluation of plasma eosinophil count and mean platelet volume in patients with coronary slow flow. Clinics. 2014;69(5):323-6.
  • Seyyed-Mohammadzad MH, et al. Slow Coronary Flow Phenomenon and Increased Platelet Volume Indices. Korean circulation journal. 2014;44(6):400-5.
  • Ucgun T, et al. Serum visfatin and omentin levels in slow coronary flow. Revista Portuguesa de Cardiologia. 2014;33(12):789-94
  • Durakoğlugil ME, et al. Increased circulating soluble CD40 levels in patients with slow coronary flow phenomenon: an observational study. Anadolu Kardiyol Derg., 2013;13(1):39-44
  • Amasyali B, et al. Aborted sudden cardiac death in a 20-year-old man with slow coronary flow. International journal of cardiology. 2006;109(3):427-9
  • Saya S, et al. Coronary slow flow phenomenon and risk for sudden cardiac death due to ventricular arrhythmias: a case report and review of literature. Clinical cardiology. 2008;31(8):352-5.
  • Karaman K, et al. New Markers for Ventricular Repolarization in Coronary Slow Flow: Tp-e Interval, Tp-e/QT Ratio, and Tp-e/QTc Ratio. Annals of Noninvasive Electrocardiology. 2014;1(1):1-7
  • Surgit O, et al. The Effect of Slow Coronary Artery Flow on Microvolt T-Wave Alternans. Acta Cardiologica Sinica. 2014;30(3):190-6
  • Kuchar DL, et al. Late potentials on the signal-averaged electrocardiogram after canine myocardial infarction: Correlation with induced ventricular arrhythmias during the healing phase. Journal of the American College of Cardiology. 1990;15(6): 1365-73
  • De Bakker J, et al. Reentry as a cause of ventricular tachycardia in patients with chronic ischemic heart disease: electrophysiologic and anatomic correlation. Circulation. 1988;77(3):589-606
  • Pogwizd SM, et al. Reentrant and focal mechanisms underlying ventricular tachycardia in the human heart. Circulation. 1992;86(6):1872-87
  • McGuire M, et al. Natural history of late potentials in the first ten days after acute myocardial infarction and relation to early ventricular arrhythmias. The American journal of cardiology. 1988;61(15):1187-90
  • Kuchar DL, Thorburn CW, Sammel NL. Late potentials detected after myocardial infarction: natural history and prognostic significance. Circulation. 1986;74(6): 1280-9
  • Denniss AR, et al. Changes in ventricular activation time on the signal-averaged electrocardiogram in the first year after acute myocardial infarction. The American journal of cardiology. 1987;60(7):580-3
  • Steinberg JS, et al. Predicting arrhythmic events after acute myocardial infarction using the signal-averaged electrocardiogram. The American journal of cardiology. 1992; 69(1):13-21
  • Simson MB. Use of signals in the terminal QRS complex to identify patients with ventricular tachycardia after myocardial infarction. Circulation. 1981;64(2):235-42
  • Breithardt G, et al. Standards for analysis of ventricular late potentials using high-resolution or signal-averaged electrocardiography: a statement by a task force committee of the European Society of Cardiology, the American Heart Association, and the American College of Cardiology. Journal of the American College of Cardiology. 1991;17(5):999-1006
  • Association AP. Diagnostic and statistical manual of mental disorders: DSM-IV-TR®. 2000: American Psychiatric Pub
  • Gibson CM, et al. TIMI frame count a quantitative method of assessing coronary artery flow. Circulation. 1996;93(5):879-88
  • Kalay N, et al. The relationship between inflammation and slow coronary flow: increased red cell distribution width and serum uric acid levels. Turk Kardiyol Dern Ars. 2011;39(6):463-8.
  • Yildirim M, et al. Thrombin activatable fibrinolysis inhibitor. Herz. 2013. p. 1-8
  • Turhan H, Yetkin E. The relation between insulin resistance and slow coronary flow: The development of microvascular dysfunction in insulin resistant state may be a plausible explanation. International journal of cardiology. 2006;111(3):474-5.
  • Elsherbiny IA, Shoukry A, Tahlawi MAE. Mean platelet volume and its relation to insulin resistance in non-diabetic patients with slow coronary flow. Journal of cardiology. 2012;59(2):176-81
  • Cain ME, et al. Signal-averaged electrocardiography. Journal of the American College of Cardiology. 1996;27(1):238-49.
  • Denniss AR, et al. Differences between patients with ventricular tachycardia and ventricular fibrillation as assessed by signal-averaged electrocardiogram, radionuclide ventriculography and cardiac mapping. Journal of the American College of Cardiology. 1988;11(2):276-83
  • Martinez-Rubio A, et al. Electrophysiologic variables characterizing the induction of ventricular tachycardia versus ventricular fibrillation after myocardial infarction: relation between ventricular late potentials and coupling intervals for the induction of sustained ventricular tachyarrhythmias. Journal of the American College of Cardiology. 1993;21(7):1624-31.
  • Lindsay BD, et al. Noninvasive detection of patients with ischemic and nonischemic heart disease prone to ventricular fibrillation. Journal of the American College of Cardiology. 1990;16(7):1656-64.
  • Beltrame JF, et al. The angiographic and clinical benefits of mibefradil in the coronary slow flow phenomenon. Journal of the American College of Cardiology. 2004; 44(1):57-62
Еще
Статья научная