С-Jun N-терминальные киназы и их модуляторы при ишемически-реперфузионном повреждении миокарда (обзор литературы)

Автор: Шведова Мария Витальевна, Анфиногенова Яна Джоновна, Попов Сергей Валентинович, Щепеткин Игорь Александрович, Аточин Дмитрий Николаевич

Журнал: Сибирский журнал клинической и экспериментальной медицины @cardiotomsk

Рубрика: Обзоры и лекции

Статья в выпуске: 3 т.31, 2016 года.

Бесплатный доступ

Короткий адрес: https://sciup.org/14920293

IDR: 14920293

Список литературы С-Jun N-терминальные киназы и их модуляторы при ишемически-реперфузионном повреждении миокарда (обзор литературы)

  • Влаопулос С., Зумпурлис В.С. JNK: ключевой модулятор внутриклеточной сигнальной системы//Биохимия. -2004. -№ 69(8). -С. 1038-1050.
  • Зюзьков Г.Н., Жданов В.В., Удут Е.В. и др. Роль JNK и участие p53 в реализации ростового потенциала мезенхимных клеток-предшественников в условиях in vitro//Бюллетень экспериментальной биологии и медицины. -2015. -№ 159(2). -С. 205-208.
  • Маслов Л.Н., Мрочек А.Г., Щепёткин И.А. и др. Роль протеинкиназ в формировании адаптивного феномена ишемического посткондиционирования сердца//Рос. физиологический журнал им. И.М. Сеченова. -2013. -№ 99(4). -С. 433-452.
  • Рязанцева Н.В., Новицкий В.В., Часовских Н.Ю. и др. Роль редокс-зависимых сигнальных систем в регуляции апоптоза при окислительном стрессе//Цитология. -2009. -№ 51(4). -С. 329-334.
  • Aoki H., Kang P.M., Hampe J. et al. Direct activation of mitochondrial apoptosis machinery by c-Jun N-terminal kinase in adult cardiac myocytes//J. Biol. Chem. -2002. -Vol. 277(12). -P. 10244-10250.
  • Armstrong S.C. Protein kinase activation and myocardial ischemia/reperfusion injury//Cardiovasc. Res. -2004. -Vol. 61(3). -P. 427-436.
  • Arslan F., Lai R.C., Smeets M.B. et al. Mesenchymal stem cell-derived exosomes increase ATP levels, decrease oxidative stress and activate PI3K/Akt pathway to enhance myocardial viability and prevent adverse remodeling after myocardial ischemia/reperfusion injury//Stem Cell Res. -2013. -Vol. 10(3). -P. 301-312.
  • Atochin D.N., Schepetkin I.A., Khlebnikov A.I. et al. A novel dual NO-donating oxime and c-Jun N-terminal kinase inhibitor protects against cerebral ischemia-reperfusion injury in mice//Neurosci. Lett. -2016. -Vol. 618. -P. 45-49.
  • Barancik M., Htun P., Schaper W. Okadaic acid and anisomycin are protective and stimulate the SAPK/JNK pathway//J. Cardiovasc. Pharmacol. -1999. -Vol. 34(2). -P. 182-190.
  • Becatti M., Taddei N., Cecchi C. et al. SIRT1 modulates MAPK pathways in ischemic-reperfused cardiomyocytes//Cell. Mol. Life Sci. -2012. -Vol. 69(13). -P. 2245-2260.
  • Bode A.M., Dong Z. The functional contrariety of JNK//Mol. Carcinog. -2007. -Vol. 46(8). -P. 591-598.
  • Bogoyevitch M.A., Kobe B. Uses for JNK: the many and varied substrates of the c-Jun N-terminal kinases//Microbiol. Mol. Biol. Rev. -2006. -Vol. 70(4). -P. 1061-1095.
  • Chambers J.W., Pachori A., Howard S. et al. Inhibition of JNK mitochondrial localization and signaling is protective against ischemia/reperfusion injury in rats//J. Biol. Chem. -2013. -Vol. 288(6). -P. 4000-4011.
  • Chaudhury H., Zakkar M., Boyle J. et al. C-Jun N-terminal kinase primes endothelial cells at atheroprone sites for apoptosis//Arterioscler. Thromb. Vasc. Biol. -2010. -Vol. 30(3). -P. 546-553.
  • Chen Y.C., Jinn T.R., Chung T.Y. et al. Magnesium lithospermate B extracted from Salvia miltiorrhiza elevates intracellular Ca2+ level in SH-SY5Y cells//Acta Pharmacol. Sin. -2010. -Vol. 31(8). -P. 923-929.
  • Clerk A., Fuller S.J., Michael A. et al. Stimulation of "stress-regulated" mitogen-activated protein kinases (stress-activated protein kinases/c-Jun N-terminal kinases and p38-mitogen-activated protein kinases) in perfused rat hearts by oxidative and other stresses//J. Biol. Chem. -1998. -Vol. 273(13). -P. 7228-7234.
  • Dougherty C.J., Kubasiak L.A., Frazier D.P. et al. Mitochondrial signals initiate the activation of c-Jun N-terminal kinase (JNK) by hypoxia-reoxygenation//FASEB J. -2004. -Vol. 18(10). -P. 1060-1070.
  • Dougherty C.J., Kubasiak L.A., Prentice H. et al. Activation of c-Jun N-terminal kinase promotes survival of cardiac myocytes after oxidative stress//Biochem. J. -2002. -Vol. 362 (Pt. 3). -P. 561-571.
  • Duplain H. Salvage of ischemic myocardium: a focus on JNK//Adv. Exp. Med. Biol. -2006. -Vol. 588. -P. 157-164.
  • Engelbrecht A.M., Niesler C., Page C. et al. P38 and JNK have distinct regulatory functions on the development of apoptosis during simulated ischaemia and reperfusion in neonatal cardiomyocytes//Basic Res. Cardiol. -2004. -Vol. 99(5). -P. 338-350.
  • Ferrandi C., Ballerio R., Gaillard P. et al. Inhibition of c-Jun N-terminal kinase decreases cardiomyocyte apoptosis and infarct size after myocardial ischemia and reperfusion in anaesthetized rats//Br. J. Pharmacol. -2004. -Vol. 142(6). -P. 953-960.
  • Frazier D.P., Wilson A., Dougherty C.J. et al. PKC-alpha and TAK-1 are intermediates in the activation of c-Jun NH2-terminal kinase by hypoxia-reoxygenation//Am. J. Physiol. Heart Circ. Physiol. -2007. -Vol. 292(4). -P. H1675-1684.
  • Fryer R.M., Patel H.H., Hsu A.K. et al. Stress-activated protein kinase phosphorylation during cardioprotection in the ischemic myocardium//Am. J. Physiol. Heart. Circ. Physiol. -2001. -Vol. 281(3). -P. H1184-1192.
  • Gehringer M., Muth F., Koch P., Laufer S.A. c-Jun N-terminal kinase inhibitors: a patent review (2010-2014)//Expert Opin. Ther. Pat. -2015. -Vol. 25(8). -P. 849-872.
  • Gupta S., Barrett T., Whitmarsh A.J. et al. Selective interaction of JNK protein kinase isoforms with transcription factors//The EMBO Journal. -1996. -Vol. 15(11). -P. 2760-2770.
  • Hausenloy D.J., Yellon D.M. Survival kinases in ischemic preconditioning and postconditioning//Cardiovasc. Res. -2006. -Vol. 70(2). -P. 240-253.
  • Hausenloy D.J., Yellon D.M. Preconditioning and postconditioning: united at reperfusion//Pharmacol. Ther. -2007. -Vol. 116(2). -P. 173-191.
  • He H., Li H.L., Lin A. et al. Activation of the JNK pathway is important for cardiomyocyte death in response to simulated ischemia//Cell Death Differ. -1999. -Vol. 6(10). -P. 987-991.
  • Hreniuk D., Garay M., Gaarde W. et al. Inhibition of c-Jun N-terminal kinase 1, but not c-Jun N-terminal kinase 2, suppresses apoptosis induced by ischemia/reoxygenation in rat cardiac myocytes//Mol. Pharmacol. -2001. -Vol. 59(4). -P. 867-874.
  • Ip Y.T., Davis R.J. Signal transduction by the c-Jun N-terminal kinase (JNK) -from inflammation to development//Curr. Opin. Cell Biol. -1998. -Vol. 10(2). -P. 205-219.
  • Irving E.A., Bamford M. Role of mitogen-and stress-activated kinases in ischemic injury//J. Cereb. Blood Flow Metab. -2002. -Vol. 22(6). -P. 631-647.
  • Jang S., Javadov S. Inhibition of JNK aggravates the recovery of rat hearts after global ischemia: the role of mitochondrial JNK//PLoS One. -2014. -Vol. 9(11). -P. e113526.
  • Javadov S., Jang S., Agostini B. Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectives//Pharmacol. Ther. -2014. -Vol. 144(2). -P. 202-225.
  • Johnson G.L., Nakamura K. The c-jun kinase/stress-activated pathway: regulation, function and role in human disease//Biochim. Biophys. Acta. -2007. -Vol. 1773(8). -P. 1341-1348.
  • Kaiser R.A., Liang Q., Bueno O. et al. Genetic inhibition or activation of JNK1/2 protects the myocardium from ischemia-reperfusion-induced cell death in vivo//J. Biol. Chem. -2005. -Vol. 280(38). -P. 32602-32608.
  • Khalid S., Drasche A., Thurner M. et al. c-Jun N-terminal kinase (JNK) phosphorylation of serine 36 is critical for p66Shc activation//Sci. Rep. -2016. -Vol. 6. -P. 20930.
  • Khandoudi N., Delerive P., Berrebi-Bertrand I. et al. Rosiglitazone, a peroxisome proliferator-activated receptor-gamma, inhibits the Jun NH(2)-terminal kinase/activating protein 1 pathway and protects the heart from ischemia/reperfusion injury//Diabetes. -2002. -Vol. 51(5). -P. 1507-1514.
  • Knight R.J., Buxton D.B. Stimulation of c-Jun kinase and mitogen-activated protein kinase by ischemia and reperfusion in the perfused rat heart//Biochem. Biophys. Res. Commun. -1996. -Vol. 218(1). -P. 83-88.
  • Laderoute K.R., Webster K.A. Hypoxia/reoxygenation stimulates Jun kinase activity through redox signaling in cardiac myocytes//Circ. Res. -1997. -Vol. 80(3). -P. 336-344.
  • Li H.H., Du J., Fan Y.N. et al. The ubiquitin ligase MuRF1 protects against cardiac ischemia/reperfusion injury by its proteasome-dependent degradation of phospho-c-Jun//Am. J. Pathol. -2011. -Vol. 178(3). -P. 1043-1058.
  • Li C., Gao Y., Tian J. et al. Sophocarpine administration preserves myocardial function from ischemia-reperfusion in rats via NF-кB inactivation//J. Ethnopharmacol. -2011. -Vol. 135(3). -P. 620-625.
  • Li X.M., Ma Y.T., Yang Y.N. et al. Ischemic postconditioning protects hypertrophic myocardium by ERK1/2 signaling pathway: experiment with mice//Zhonghua Yi Xue Za Zhi. -2009. -Vol. 89(12). -P. 846-850.
  • Li C., Wang T., Zhang C. et al. Quercetin attenuates cardiomyocyte apoptosis via inhibition of JNK and p38 mitogen-activated protein kinase signaling pathways//Gene. -2016. -Vol. 577(2). -P. 275-280.
  • Liu Q., Wang J., Liang Q. et al. Sparstolonin B attenuates hypoxia-reoxygenation-induced cardiomyocyte inflammation//Exp. Biol. Med. (Maywood). -2014. -Vol. 239(3). -P. 376-384.
  • Liu X., Xu F., Fu Y. et al. Calreticulin induces delayed cardioprotection through mitogen-activated protein kinases//Proteomics. -2006. -Vol. 6(13). -P. 3792-3800.
  • Liu H.T., Zhang H.F., Si R. et al. Insulin protects isolated hearts from ischemia/reperfusion injury: cross-talk between PI3-K/Akt and JNKs//Acta Physiol. Sin. -2007. -Vol. 59(5). -P. 651-659.
  • Liu X.H., Zhang Z.Y., Sun S. et al. Ischemic postconditioning protects myocardium from ischemia/reperfusion injury through attenuating endoplasmic reticulum stress//Shock. -2008. -Vol. 30(4). -P. 422-427.
  • Messoussi A., Feneyrolles C., Bros A. et al. Recent progress in the design, study, and development of c-Jun N-terminal kinase inhibitors as anticancer agents//Chem. Biol. -2014. -Vol. 21(11). -P. 1433-1443.
  • Milano G., Morel S., Bonny C. et al. A peptide inhibitor of c-Jun NH2-terminal kinase reduces myocardial ischemia-reperfusion injury and infarct size in vivo//Am. J. Physiol. Heart Circ. Physiol. -2007. -Vol. 292(4). -P. H1828-1835.
  • Morrison A., Yan X., Tong C. et al. Acute rosiglitazone treatment is cardioprotective against ischemia-reperfusion injury by modulating AMPK, Akt, and JNK signaling in nondiabetic mice//Am. J. Physiol. Heart Circ. Physiol. -2011. -Vol. 301(3). -P. H895-902.
  • Nakano A., Baines C.P., Kim S.O. et al. Ischemic preconditioning activates MAPKAPK2 in the isolated rabbit heart: evidence for involvement of p38 MAPK//Circ. Res. -2000. -Vol. 86(2). -P. 144-151.
  • Nijboer C.H., van der Kooij M.A., van Bel F. et al. Inhibition of the JNK/AP-1 pathway reduces neuronal death and improves behavioral outcome after neonatal hypoxic-ischemic brain injury//Brain Behav. Immun. -2010. -Vol. 24(5). -P. 812-821.
  • Oshikawa J., Kim S.J., Furuta E. et al. Novel role of p66Shc in ROS-dependent VEGF signaling and angiogenesis in endothelial cells//Am. J. Physiol. Heart Circ. Physiol. -2012. -Vol. 302(3). -P. H724-732.
  • Ping P., Zhang J., Huang S. et al. PKC-dependent activation of p46/p54 JNKs during ischemic preconditioning in conscious rabbits//Am. J. Physiol. -1999. -Vol. 277(5 Pt. 2). -P. H1771-1785.
  • Qi D., Hu X., Wu X. et al. Cardiac macrophage migration inhibitory factor inhibits JNK pathway activation and injury during ischemia/reperfusion//J. Clin. Invest. -2009. -Vol. 119(12). -P. 3807-3816.
  • Rose B.A., Force T., Wang Y. Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale//Physiol. Rev. -2010. -Vol. 90(4). -P. 1507-1546.
  • Sato M., Bagchi D., Tosaki A. et al. Grape seed proanthocyanidin reduces cardiomyocyte apoptosis by inhibiting ischemia/reperfusion-induced activation of JNK-1 and C-JUN//Free Radic. Biol. Med. -2001. -Vol. 31(6). -P. 729-737.
  • Shang L., Ananthakrishnan R., Li Q. et al. RAGE modulates hypoxia/reoxygenation injury in adult murine cardiomyocytes via JNK and GSK-3beta signaling pathways//PLoS One. -2010. -Vol. 5(4). -P. e10092.
  • Shao Z., Bhattacharya K., Hsich E. et al. c-Jun N-terminal kinases mediate reactivation of Akt and cardiomyocyte survival after hypoxic injury in vitro and in vivo//Circ. Res. -2006. -Vol. 98(1). -P. 111-118.
  • Shi S., Li Q.S., Li H. et al. Anti-apoptotic action of hydrogen sulfide is associated with early JNK inhibition//Cell Biol. Int. -2009. -Vol. 33(10). -P. 1095-1101.
  • Song Z.F., Ji X.P., Li X.X. et al. Inhibition of the activity of poly (ADP-ribose) polymerase reduces heart ischaemia/reperfusion injury via suppressing JNK-mediated AIF translocation//Cell Mol. Med. -2008. -Vol. 12(4). -P. 1220-1228.
  • Sun L., Isaak C.K., Zhou Y. et al. Salidroside and tyrosol from Rhodiola protect H9c2 cells from ischemia/reperfusion-induced apoptosis//Life Sci. -2012. -Vol. 91(5-6). -P. 151-158.
  • Sun H.Y., Wang N.P., Halkos M. et al. Postconditioning attenuates cardiomyocyte Apoptosis via inhibition of JNK and p38 mitogen-activated protein kinase signaling pathways//Apoptosis. -2006. -Vol. 11(9). -P. 1583-1593.
  • Talmor D., Applebaum A., Rudich A. et al. Activation of mitogen-activated protein kinases in human heart during cardiopulmonary bypass//Circ. Res. -2000. -Vol. 86(9). -P. 1004-1007.
  • Vassalli G., Milano G., Moccetti T. Role of Mitogen-Activated Protein Kinases in myocardial ischemia-reperfusion injury during heart transplantation//J. Transplant. -2012. -Vol. 2012. -P. 928954.
  • Waetzig V., Herdegen T. Context-specific inhibition of JNKs: overcoming the dilemma of protection and damage//Trends Pharmacol. Sci. -2005. -Vol. 26(9). -P. 455-461.
  • Walshe C.M., Laffey J.G., Kevin L. et al. Sepsis protects the myocardium and other organs from subsequent ischaemic/reperfusion injury via a MAPK-dependent mechanism//Intensive Care Med. Exp. -2015. -Vol. 3(1). -P. 35.
  • Wang Z., Huang H., He W. et al. Regulator of G-protein signaling 5 protects cardiomyocytes against apoptosis during in vitro cardiac ischemia-reperfusion in mice by inhibiting both JNK and P38 signaling pathways //Biochem. Biophys. Res. Commun. -2016. -Vol. 473(2). -P. 551-557.
  • Wang J., Yang L., Rezaie A.R. et al. Activated protein C protects against myocardial ischemic/reperfusion injury through AMP-activated protein kinase signaling//J. Thromb. Haemost. -2011. -Vol. 9(7). -P. 1308-1317.
  • Wei J., Wang W., Chopra I. et al. C-Jun N-terminal kinase (JNK-1) confers protection against brief but not extended ischemia during acute myocardial infarction//J. Biol. Chem. -2011. -Vol. 286(16). -P. 13995-14006.
  • Wei C., Zhao Y., Wang L. et al. H2S restores the cardioprotection from ischemic post-conditioning in isolated aged rat hearts//Cell Biol. Int. -2015. -Vol. 39(10). -P. 1173-1176.
  • Wiltshire C., Gillespie D.A., May G.H. Sab (SH3BP5), a novel mitochondria-localized JNK-interacting protein//Biochem. Soc. Trans. -2004. -Vol. 32 (Pt. 6). -P. 1075-1077.
  • Wu J., Li J., Zhang N. et al. Stem cell-based therapies in ischemic heart diseases: a focus on aspects of microcirculation and inflammation//Basic Res. Cardiol. -2011. -Vol. 106(3). -P. 317-324.
  • Xie P., Guo S., Fan Y. et al. Atrogin-1/MAFbx enhances simulated ischemia/reperfusion-induced apoptosis in cardiomyocytes through degradation of MAPK phosphatase-1 and sustained JNK activation//J. Biol. Chem. -2009. -Vol. 284(9). -P. 5488-5496.
  • Xu J., Qin X., Cai X. et al. Mitochondrial JNK activation triggers autophagy and apoptosis and aggravates myocardial injury following ischemia/reperfusion//Biochim. Biophys. Acta. -2015. -Vol. 1852(2). -P. 262-270.
  • Xu T., Wu X., Chen Q. et al. The anti-apoptotic and cardioprotective effects of salvianolic acid A on rat cardiomyocytes following ischemia/reperfusion by DUSP-mediated regulation of the ERK1/2/JNK pathway//PLoS One. -2014. -Vol. 9(7). -P. e102292.
  • Xu H., Yao Y., Su Z. et al. Endogenous HMGB1 contributes to ischemia-reperfusion-induced myocardial apoptosis by potentiating the effect of TNF-alpha/JNK//Am. J. Physiol. Heart Circ. Physiol. -2011. -Vol. 300(3). -P. H913-921.
  • Yang L.M., Xiao Y.L., Ou-Yang J.H. Inhibition of magnesium lithospermate B on the c-Jun N-terminal kinase 3 mRNA expression in cardiomyocytes encountered ischemia/reperfusion injury//Acta Pharmacol. Sin. -2003. -Vol. 38(7). -P. 487-491.
  • Yin T., Sandhu G., Wolfgang C.D. et al. Tissue-specific pattern of stress kinase activation in ischemic/reperfused heart and kidney//J. Biol. Chem. -1997. -Vol. 272(32). -P. 19943-19950.
  • Yue T.L., Wang C., Gu J.L. et al. Inhibition of extracellular signal-regulated kinase enhances ischemia/reoxygenation-induced apoptosis in cultured cardiac myocytes and exaggerates reperfusion injury in isolated perfused heart//Circ. Res. -2000. -Vol. 86(6). -P. 692-699.
  • Zaha V.G., Qi D., Su K.N. et al. AMPK is critical for mitochondrial function during reperfusion after myocardial ischemia//J. Mol. Cell. Cardiol. -2016. -Vol. 91. -P. 104-113.
  • Zhang J., Li X.X., Bian H.J. et al. Inhibition of the activity of Rho-kinase reduces cardiomyocyte apoptosis in heart ischemia/reperfusion via suppressing JNK-mediated AIF translocation//Clin. Chim. Acta. -2009. -Vol. 401(1-2). -P. 76-80.
  • Zhang G.M., Wang Y., Li T.D. et al. Change of JNK MAPK and its influence on cardiocyte apoptosis in ischemic postconditioning//J. Zhejiang Univ. -2009. -Vol. 38(6). -P. 611-619.
  • Zhang G.M., Wang Y., Li T.D. et al. Post-conditioning with gradually increased reperfusion provides better cardioprotection in rats//World J. Emerg. Med. -2014. -Vol. 5(2). -P. 128-134.
  • Zinkel S., Gross A., Yang E. BCL2 family in DNA damage and cell cycle control//Cell Death Differ. -2006. -Vol. 13(8). -P. 1351-1359.
Еще
Статья